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and experiments with the co-administration of AIs and pro-algesic stimuli, such as PAR2-AP, an agonist of the pro-inflammatory receptor, PAR2, and the TRPA1 agonist, H2O2 (ref

and experiments with the co-administration of AIs and pro-algesic stimuli, such as PAR2-AP, an agonist of the pro-inflammatory receptor, PAR2, and the TRPA1 agonist, H2O2 (ref. decreased grip strength, which do not undergo desensitization on long term AI administration. These effects Eugenol are markedly attenuated by TRPA1 pharmacological blockade or in TRPA1-deficient mice. TRPA1 is […]